Addiction and Withdrawal Restoration
- 10 Day iv infusion (recommended for instant recovery)
- Minimal Withdrawal after 5th day.
- Restores brain cells back to pre-addiction condition
- Works best for Opiate and Alcohol Addiction
- This program is self pay only
SPECIAL OFFER !!
10 DAY ADDICTION PROGRAM –
$7,900.00 NOW $5,800.00
4 DAY REVERSE AGING PROGRAM –
$5,800.00 NOW $3,800.00
What is NAD?
NAD or Nicotinamide Adenine Dinucleotide is a simple metabolic coenzyme of Niacin. Niacin is a B Vitamin that is involved in energy production in every mitochondria of your body. Niacin is the key fuel for energy production in every cell of the body, administered in an iv form, is clinically proven with a 90% no-craving statistic, Nad helps people detox off of alcohol, opiates, stimulants and tranquilizers. It is also effective in treating depression, anxiety and PTSD.
Nicotinamide Adenine Dinucleotide (NAD) relieves withdrawal symptoms, flushes out stored drugs in the body and replenishes balance in the brain.
HOW THE TREATMENT WORKS
The scientific explanation behind the Brain Restoration treatment and the proprietary NAD formula administered by iv. is an element that reacts with oxygen in the cell’s mitochondria in order to create energy for movement, breathing, heartbeat, blood pumping, digesting food, brain functions, and generally living life. Studies have found that those with extremely low NAD levels (which can be present even at birth) are far more vulnerable to addiction as well as other diseases and to chronic physical conditions. There is a preponderance of low levels of NAD present in Western society as it is mostly lost in cooking and food processing. What little remains is broken down by stomach acid, degraded before it’s absorbed from the digestive tract.
iv INFUSED (NAD)
When NAD is received directly through an iv, the nutrients bypass the stomach and go directly to the receptors in the brain, this immediately produces palpable positive results as the nutrients bathe the brain in a continuous pool of natural and highly therapeutic co-enzymes. Since NAD is a detoxifier, it takes days (rather than weeks or months), to flush out stored drugs from the body and its organs, replenish balance in the brain, and reverse damage. Results can be mental clarity, cognitive function increase, focus and concentration returns, more energy, better mood, and positive outlook. And this happens cold turkey.
Research has shown that NAD increases the synthesis of certain neurotransmitters in the brain known to be effective in correcting specific chemical imbalances. Some of these chemical imbalances underpin addiction, mental illness, anxiety, aggression, depression, despair and hopelessness. Fatigue is often the first signal of NAD deprivation; other clues may include depression and anxiety in children. Almost any chronic disease, including Parkinson’s, can also be indicative of deficiency.
There is some research and other reports indicating that NAD might be effective treatment for a host of other ailments including schizophrenia, PTSD, chronic fatigue, weak immune system, memory disturbance, sleep problems, concentration defects, blood pressure, poor cholesterol levels, sugar metabolism and diabetes, muscle pain and weakness, joint pain and stiffness, headaches, fevers, sore throats and swollen lymph glands. Clinical research has shown it is a potent biological antioxidant, which can aid in preventing cell damage and a variety of diseases, cancer included.
There is also some evidence that NAD therapy can help with aging. Dr. David Sinclair, professor of genetics at Harvard Medical School, in a paper published in the journal Cell, describes a compound naturally made by young cells that is able to revive older cells, allowing them to be energetic and youthful again. With adequate amounts of NAD, aging can theoretically be reversed, he asserts. “When we give the molecule, the cells think oxygen levels are normal and everything revs back up again,” Sinclair wrote.
Pondering these claims raises the un-researched theory of whether NAD deficiency might be an unrecognized epidemic disease of our time.
The coenzyme NAD+ was first discovered by the British biochemists Arthur Harden and William John Young in 1906. They noticed that adding boiled and filtered yeast extract greatly accelerated alcoholic fermentation in unboiled yeast extracts. They called the unidentified factor responsible for this effect a coferment. Through a long and difficult purification from yeast extracts, this heat-stable factor was identified as a nucleotide sugar phosphate by Hans von Euler-Chelpin. In 1936, the German scientist Otto Heinrich Warburg showed the function of the nucleotide coenzyme in hydride transfer and identified the nicotinamide portion as the site of redox reactions.
Vitamin precursors of NAD+ were first identified in 1938, when Conrad Elvehjem showed that liver has an “anti-black tongue” activity in the form of nicotinamide. Then, in 1939, he provided the first strong evidence that niacin is used to synthesize NAD+. In the early 1940s, Arthur Kornberg made another important contribution towards understanding NAD+ metabolism, by being the first to detect an enzyme in the biosynthetic pathway. Subsequently, in 1949, the American biochemists Morris Friedkin and Albert L. Lehninger proved that NADH linked metabolic pathways such as the citric acid cycle with the synthesis of ATP in oxidative phosphorylation. In 1958, Jack Preiss and Philip Handler discovered the intermediates and enzymes involved in the biosynthesis of NAD+[ consequently, salvage synthesis from nicotinic acid is termed the Preiss-Handler pathway. In 2004, Charles Brennerand co-workers uncovered the nicotinamide riboside kinase pathway to NAD+.
The non-redox roles of NAD(P) are a recent discovery. The first of these functions to be identified was the use of NAD+ as the ADP-ribose donor in ADP-ribosylation reactions, observed in the early 1960s Later studies in the 1980s and 1990s revealed the activities of NAD+ and NADP+ metabolites in cell signaling – such as the action of cyclic ADP-ribose, which was discovered in 1987. The metabolism of NAD+has remained an area of intense research into the 21st century, with interest being heightened after the discovery of the NAD+-dependent protein deacetylases calledsirtuins in 2000, by Shin-ichiro Imai and coworkers at the Massachusetts Institute of Technology.
PLoS One. 2012;7(10):e44933. doi: 10.1371/journal.pone.0044933. Epub 2012 Oct 11.
Cell Metab. 2011 Oct 5;14(4):528-36. doi: 10.1016/j.cmet.2011.08.014.
J Nutr Biochem. 2014 Jan;25(1):66-72. doi: 10.1016/j.jnutbio.2013.09.004. Epub 2013 Oct 10.
Mol Cells. 2013 Jun;35(6):474-80. doi: 10.1007/s10059-013-0130-x. Epub 2013 May 8.
Crit Rev Biochem Mol Biol. 2013 Jul-Aug;48(4):397-408. doi: 10.3109/10409238.2013.789479. Epub 2013 Jun 6.
J Reprod Dev. 2013;59(3):238-44. Epub 2013 Mar 10.
Curr Opin Clin Nutr Metab Care. 2013 Nov;16(6):657-61. doi: 10.1097/MCO.0b013e32836510c0.
Neurobiol Aging. 2013 Jun;34(6):1581-8. doi: 10.1016/j.neurobiolaging.2012.12.005. Epub 2013 Jan 9.
Nicotinamide riboside restores cognition through an upregulation of proliferator-activated receptor-γ coactivator 1α regulated β-secretase 1 degradation and mitochondrial gene expression in Alzheimer’s mouse models.
Neurobiol Aging. 2013 Jun;34(6):1564-80. doi: 10.1016/j.neurobiolaging.2012.11.020. Epub 2012 Dec 25.
Rev Clin Esp. 2010 Jun;210(6):284-8. doi: 10.1016/j.rce.2009.09.015. Epub 2010 May 5.
Rejuvenation Res. 2010 Apr-Jun;13(2-3):159-61. doi: 10.1089/rej.2009.0918.